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You perhaps haven’t been reading any of the papers coming out of Europe?

In my submission history you can see just yesterday an MRI study out of Europe. 78% of patients had cardiovascular involvement, 60% of patients still had heart inflammation.

These were 100 Germans, tested 2-3 months after diagnosis. And 2/3rd of the cases in the study were mild at home cases, no hospitalization. There was no large difference in heart involvement between groups.

A fairly large amount of the literature on these effects is actually from Europe as you got hit a month before the US, so there’s been more time to write studies.

I would not say you have any evidence in support of your position. I certainly have not heard of any studies finding covid to be a multi fanged beast in america and a mere respiratory virus in the rest of the world.

Edit: the paper: https://jamanetwork.com/journals/jamacardiology/fullarticle/...



I doubt the virus is still around 2-3 months later in their bodies. These effects are likely due to the hyperinflammation triggered later on during the course of the disease, if I am to follow the mechanism described in the well-detailed paper in Cell from a few weeks ago.

The virus is still a coronavirus (neural invasion, also noted for SARS-CoV-2, is also a feature of regular coronaviruses). These effects, although possibly severe and worthy of concern, are second-order effects.


"the virus is still a coronavirus"

That's always something i wonder every time i read about covid-19 : are we sure that those effects aren't also observed with other coronaviruses, maybe on a different intensity, but we just never had so much scrutiny for thoses virus as we have on this one in particular ?

Diarrheas etc are common symptoms of many viral infections, and people die from side effects of the flu or the cold every year ( old people most of the time).

Is the difference qualitative or quantitative ?


I can say for sure on neural invasion because I found a paper describing the phenomenon from 2018, which described (IIRC, on hospitalized patients) neural invasions by "human coronavirus".

Took a while to dig through my post history, but here's the paper I mentioned:

https://www.frontiersin.org/articles/10.3389/fncel.2018.0038...


It's somewhat similar to the mutant behavior of FCoV, which can cause feline infectious peritonitis. Regular FCoV causes some gastrointestinal and respiratory issues, but upon mutation starts breaking down cells and causing issues in the circulatory system, ultimately causing death.

https://www.sheltermedicine.com/library/resources/?r=feline-...


I agree, I do not think the heart inflammation suggests active virus. But my point was to show the commentor I replied to that the effects in Europe are not limited to respiratory effects.

They were under the impression only Americans had widespread non-respiratory symptoms of coronavirus.




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